By Angela Kucia, Tom Quinn
Acute Cardiac Care presents nurses with a complete knowing of the present perform and ideas underlying the care and administration of acute cardiac stipulations. It addresses the administration of acute coronary syndromes (ACS) with an emphasis on evidence-based pharmacological administration, cardiac emergencies (cardiac arrest, arrhythmia, acute pulmonary oedema, cardiogenic shock), present diagnostic and interventional modalities for the administration of ACS, together with chance stratification, and the moral, political, social and monetary components that impression upon the supply of acute cardiac prone in Australia and the UK.
Edited by way of popular lecturers and clinicians within the box, this functional textual content will motivate nurses to imagine severely approximately proof and administration of acute coronary stipulations, and supply a consultant on how and the place to appear for up to date proof and guidelines.
A entire and sensible advisor to the present perform and ideas underlying the nursing care of acute cardiac conditions
Edited by way of prime experts within the box with a wealth of expertise in acute cardiac care and resuscitation
Has an emphasis on evidence-based perform, encouraging nurses to significantly take into consideration their practice
Contains studying goals, key issues and actions and additional interpreting guidelines
Read Online or Download Acute Cardiac Care: A Practical Guide for Nurses PDF
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Extra info for Acute Cardiac Care: A Practical Guide for Nurses
Caϩϩ channels close and Kϩ ﬂows rapidly out of the cell. Phase 4 refers to the phase where the cell is not stimulated (the resting membrane potential). This phase coincides with diastole. Kϩ is restored to the inside of the cell and Naϩ to the outside by active transport through the sodium–potassium pump. Key point The ability of the cell to open fast Naϩ channels during phase 0 is related to the membrane potential at the moment of excitation. For the cell to be able to open the fast Naϩ channels, the resting membrane potential must be at baseline (85– 90 mV) and all Naϩ channels closed.
1). ● Phase 0 represents the rapid depolarisation phase where the fast sodium channels open and there is a rapid inﬂux of Naϩ into the cell. Calcium moves slowly but steadily into the cell. The membrane potential moves from the negative charge of 85–90 mV to ϩ10–20 mV. This creates a gradient with the surrounding cell membranes, allowing the electrical current to ﬂow from the depolarised cell to the surrounding cells, propagating the impulse. Phase 1 represents an initial repolarisation of the cell caused by opening of special transient outward Kϩ channels and the inactivation of the Naϩ channels.
Both type 1 and type 2 diabetes mellitus are independent risk factors for CVD (McGill & McMahan 1998; Wilson et al. 5). 5 with the common clustering of these risk factors in a single individual known as the metabolic syndrome (Grundy et al. 1999). Testing for diabetes The fasting blood glucose (FBG) and oral glucose tolerance test (OGTT), also referred to as the glucose tolerance test, are blood tests used in the diagnosis of diabetes. The OGTT measures the body’s ability to metabolise glucose, or clear it out of the bloodstream, and although it is more time consuming than the FBG, it is a more sensitive measure and can be used to diagnose diabetes, gestational diabetes (diabetes during pregnancy) or pre-diabetes.